2001-
Lopatin AN and Nichols CG. Inward rectifiers in the heart: An update on IK1.
Journal of Molecular and Cellular Cardiology, 2001;
33(4): 625-638
Lopatin AN Inward rectification and cardiac excitability. Biological
Membranes (Russ), 2002; 19(1): 57-65
McLerie
M and Lopatin AN. Dominant negative suppression of IK1 in the
mouse heart leads to altered cardiac excitability. Journal of Molecular and
Cellular Cardiology, 2003;
35(4): 367-378
This paper
is highlighted in the Editorial. `When
less is more: what genetic manipulation tells us about cardiac I(K1).`
Journal of Molecular and Cellular Cardiology, 2003; 35(4): 343-345
Flagg
TP, Charpentier F, Manning-Fox J, Remedi MS, Enkvetchakul D, Lopatin A,
Koster J and Nichols C. Remodeling of excitation-contraction coupling in
transgenic mice expressing ATP-insensitive sarcolemmal KATP channels.
American Journal of Physiology. Heart and Circulation Physiology, 2004; 286(4): H1361-H1369
Li J, McLerie M and Lopatin AN. Transgenic up-regulation of IK1
in the mouse heart leads to multiple abnormalities of cardiac excitability. American
Journal of Physiology. Heart and Circulation Physiology, 2004;
287(6): H2790-H2802
Flagg
TP, Remedi MS, Masia R, Gomes J, McLerie M, Lopatin AN and Nichols CG. Transgenic overexpression of SUR1 in the
heart suppresses sarcolemmal KATP. Journal of Molecular and Cellular Cardiology, 2005; 39(4): 647-656
Panama BK and Lopatin AN.
Differential polyamine sensitivity in Kir2 channels. Journal
of Physiology, 2006; 571(2):
287-302
Dobrzynski H, Billeter R, Greener ID, Tellez JO, Chandler NJ, Flagg
TP, Nichols
CG, Lopatin AN and Boyett MR.
Expression of Kir2.1 and Kir6.2 transgenes under the control of the alpha-MHC
promoter in the sinoatrial and atrioventricular nodes in transgenic mice. Journal
of Molecular and Cellular Cardiology, 2006;
41: 855-867
Noujaim SF, Pandit
SV, Berenfeld O, Vikstrom K, Cerrone M, Mironov S, Zugermayr
M, Lopatin AN and Jalife J.
Up-regulation of the inward rectifier K+ current (IK1) in
the mouse heart accelerates and stabilizes rotors. Journal of Physiology, 2007;
578: 315-326
Fu Y, Huang X, Piao L, Lopatin AN,
and Neubig RR. Endogenous RGS proteins modulate SA and AV nodal functions in
isolated heart: implications for sick sinus syndrome and AV block. American
Journal of Physiology. Heart and Circulation Physiology, 2007;
292: H2532-H2539
Flagg
TP, Patton B, Masia R,
Mansfield
C, Lopatin AN, Yamada KA and Nichols
CG. Arrhythmia susceptibility and premature death in transgenic mice
overexpressing both SUR1 and Kir6.2
[ΔN30,K185Q]
in the heart. American Journal of Physiology. Heart and Circulation
Physiology, 2007. 293(1):
H836-845
Piao L, Li J, McLerie M and Lopatin AN. Transgenic upregulation of IK1
in the mouse heart is proarrhythmic. Basic
Research in Cardiology, 2007.
102(5): 416-428
Piao L, Li J, McLerie M and Lopatin AN. Cardiac
IK1 channels underlie early action potential shortening during
hypoxia in the mouse heart. Journal of
Molecular and Cellular Cardiology, 2007.
43(1): 27-38
This paper is highlighted in the
Editorial. `IK1
and cardiac hypoxia: after the long and short QT syndromes, what else can go
wrong with the inward rectifier K+ currents?`
Journal of Molecular and Cellular Cardiology, 2007; 43(1): 15-17
Lopez-Santiago LF, Meadows LS, Ernst SJ, Chen C, Malhotra JD, McEwen DP,
Speelman A, Noebels JL, Maier SK, Lopatin
AN, Isom LL. Sodium
channel Scn1b null mice exhibit prolonged QT and RR intervals. Journal
of Molecular and Cellular Cardiology. 2007;
43: 636-47
Panama BK, McLerie M and Lopatin AN. Heterogeneity of IK1 in the mouse heart.
American Journal of Physiology. Heart and Circulation Physiology. 2007; 293(6):H3558-67
Day
SM, Coutu P, Wang W, Herron TJ, Turner I, Shillingford M, Lacross NC, Converso
KL, Piao L, Li J, Lopatin AN, Metzger JM. Cardiac-directed parvalbumin
transgene expression in mice shows marked heart rate dependence of delayed Ca2+
buffering action. Physiological
Genomics. 2008; 33: 312-322
